Slide 34:
In the early 1980’s my wife became ill. Subsequently, I started
having mothers come in to the practice who told me that by 3 or 4 in the afternoon, they
were too tired to take care of their children. Within a few years, I started having the
mothers come in telling me that the children were too tired by 3 or 4 in the afternoon.
Characteristic of this disorder in adults is cognitive dysfunction, short-term memory
problems, decreased processing ability, abnormal sleep cycles. Think about your kids
– that’s what they have.
Now if these adults grew up normal until they were 20, 30, 40 or 50
years old, if they graduated college – most of them, as they were facetiously defined
in the late 1980’s, as having "yuppie flu" – why were they called the
"yuppie flu"? These were basically college-educated adults who all of a sudden
were dysfunctional. And nobody could say why. So, unfortunately what I learned the hard
way in our system is that if you can’t measure something, then it must be
psychiatric. That’s been perhaps one of the biggest ongoing mistakes of our research
establishment.
For some reason when I was back in medical school, we accepted that
there was this single marker, the positive ANA for lupus, and that something was wrong
with those people. But, what I’ve learned the hard way is that if we can’t
measure something wrong, it’s psychiatric. At that time I was willing to look at
amino acid work, candida – which of course, is taboo in medicine. A firm I was
working with sent me over some children from west Los Angeles who were autistic. I ran
profiles on them, these amino acid profiles. What was very strange was that their profiles
were showing similar patterns of deficiencies as these adults. By that time I had already
come to think of these adults as immune-related so the thought went through my head, what
in the world did autism have to do with the immune system?
